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section of your code, create a new variable called nailOut, with a data type of Number and a value of the nail s current Y position

provided and will likely provide better insight into the classi cation of these myopathies Unfortunately, there are no medications as yet available to these disorders However, physical and occupational therapy as well as supportive therapy for respiratory or cardiac muscle involvement can be bene cial

creates an array ranging from c[0] to c[kMaxArraySize-1]. The reference to c[kMaxArraySize] is out of bounds. g. The problem occurs in the line:

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18 1 Isaacs H, Heffron JJ, et al Central core disease A correlated genetic, histochemical, ultramicroscopic, and biochemical study J Neurol Neurosurg Psychiatry 1975; 38(12):1177 1186 2 Magee KR, Shy GM A new congenital non-progressive myopathy Brain 1956;79(4):610 621 3 Quinlivan RM, Muller CR, et al Central core disease: Clinical, pathological, and genetic features Arch Dis Child 2003;88(12):1051 1055 4 Bertini E, Bosman C, et al Neuromyopathy and restrictive cardiomyopathy with accumulation of intermediate laments: A clinical, morphological and biochemical study Acta Neuropathol (Berl) 1991;81(6):632 640 5 Cancilla PA, Kalyanaraman K, et al Familial myopathy with probable lysis of myo brils in type I bers Neurology 1971;21(6):579 585 6 Amato A, Dumitru D Hereditary Myopathies Philadelphia: Hanley & Belfus, 2002 7 Sewry CA, Muller C, et al The spectrum of pathology in central core disease Neuromuscul Disord 2002;12(10): 930 938 8 Ferreiro A, Monnier N, et al A recessive form of central core disease, transiently presenting as multi-minicore disease, is associated with a homozygous mutation in the ryanodine receptor type 1 gene Ann Neurol 2002;51(6):750 759 9 De Bleecker JL, Ertl BB, et al Patterns of abnormal protein expression in target formations and unstructured cores Neuromuscul Disord 1996;6(5):339 349 10 Mathews KD, Moore SA Multiminicore myopathy, central core disease, malignant hyperthermia susceptibility, and RYR1 mutations: One disease with many faces Arch Neurol 2004;61(1):27 29 11 Avila G Intracellular Ca2+ dynamics in malignant hyperthermia and central core disease: Established concepts, new cellular mechanisms involved Cell Calcium 2005;37(2):121 127 12 Avila G, O Connell KM, et al The pore region of the skeletal muscle ryanodine receptor is a primary locus for excitation-contraction uncoupling in central core disease J Gen Physiol 2003;121(4):277 286 13 Engel AG, Gomez MR, et al Multicore disease A recently recognized congenital myopathy associated with multifocal degeneration of muscle bers Mayo Clin Proc 1971;46(10):666 681 14 Ferreiro A, Estournet B, et al Multi-minicore disease searching for boundaries: Phenotype analysis of 38 cases Ann Neurol 2000;48(5):745 757 15 Ferreiro A, Fardeau M 80th ENMC international workshop on multi-minicore disease: 1st international MmD work-.

cPtr++ = 0;

var var var var bashAmt:Number = 15; rotateAmt:Number = -15; nailIn:Number = 470; nailOut:Number = nail_mc_y;

25 26

29 30

This line assigns the pointer variable cPtr a value of 0 (making it point to location 0 in memory) and then increments it to 1 (making it point to location 1 in memory). This code will not compile. Here s a more likely scenario:

32 33

4 At the bottom of the DEFINE EVENT HANDLERS section of your code,

*cPtr++ = 0;

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